Critical Pathway: Idiopathic Pulmonary Fibrosis Essay

2568 words - 10 pages

Introduction
Mr. B is a 65 year old Caucasian male, presenting in the ER with complaints of shortness of breath, and non-productive cough for the past 8 months. He states the shortness of breath worsens with activity and frequent “coughing spells”.
Mr. B has been treated for Pneumonia and Bronchitis over the past 8 months with oral antibiotics and steroids. Mr. B noticed slight improvement of symptoms while on medication; however, the symptoms never resolved, and seem to be progressively worsening.
Mr. B is growing concerned because he has unintentionally lost weight, he tires easily, and he is unable to preach entire sermons without getting “out of breath”. Upon examination, Velcro-like rales are auscultated in bilateral better to use after lungs.lower lungs bilaterally. When talking, the patient is noted to have rapid, shallow breathing. Slight clubbing of the fingers noted. Initial vital signs were (bold indicates abnormal values):
Height- 5 ft 8 in
Weight- 170 lbs
Blood pressure- 122/78
Pulse- 100
Respirations- 28
Oxygen Saturation- 90%
Temperature- 97.6 (oral)
Blood was drawn for routine laboratory tests, and no abnormal values were noted:no ABGs ?
Comprehensive Metabolic Panel
Potassium- 3.3 low 3.5-5.5 mEq/L not addressed in paper
Sodium- 137 135-148 mEq/L
Chloride- 102 96-112 mEq/L
Carbon Dioxide- 34 21-34 mEq/L
Glucose- 73 65-99 mg/dL
Blood Urea Nitrogen- 15 8-25 mg/dL
Creatinine- 0.9 0.6-1.5 mg/dL
Calcium- 8.3 8.0-11 mg/dL
Magnesium- 1.7 1.5-2.5 mg/dL
Phosphorous- 3.3 2.2-4.8 mg/dL
Albumin- 4.3 3.5-5.0 gm/dL
AST- 13 7-21 U/L
ALT- 11 0-30 U/L
Complete Blood Count
RBC- 4.8 4.2-5.6 M/L
WBC- 8 3.8-11.0 K/mm3
Hematocrit- 42 39-54%
Hemoglobin- 16 14-18 g/dL
Platelets- 270 150-450 U/L
MCH- 31.2 27-35 pg
MCHC 36.1 31-37%
MCV- 84 78-98 fL
Chest X-Ray revealed honeycombing and decreased lung volume. Chest CT revealed honeycombing, traction bronchiectasis, and subpleural patchy opacities. No obstructive defect is identified by pulmonary function tests; however, a notable restrictive defect is uncovered.
I. Precipitating Factors
Mr. B was able to provide information regarding his medical history. He has worked as a fulltime preacher for the past 20 years, and before that he worked as a farmer with the family business. Mr. B states that because of a deep love for farming, he has continued to work whenever he has the opportunity.
The patient reports a past medical history of mild gastroesophageal reflux disease (GERD), no past surgical history, and no regular home medications. The patient has never used illegal drugs or alcohol. He has a history of smoking for 25 years; however, he quit smoking 15 years ago.
Mr. B reports a family history of coronary heart disease (father), rheumatoid arthritis (mother), cervical cancer (sister), and an unknown lung disease (sister); Mr. B knows his sister does not have COPD, but he can’t remember the specific disease name.
II....

Find Another Essay On Critical Pathway: Idiopathic Pulmonary Fibrosis

Lung transplantation Essay

885 words - 4 pages inhibition during acute rejection episodes in lung transplant recipients may limit the development of BOS. INTRODUCTION Cessation of microvascular flow may be a key cause for chronic rejection in all solid organ transplants recipients. Preserving microvasculature health in lung allografts may be critical for preventing terminal airway fibrosis, also known as the bronchiolitis obliterans syndrome (BOS) (Babu et al., 2007), a fibrotic process

The Symptoms, Causes and Remedies for Cystic Fibrosis

1287 words - 6 pages Fibrosis patients around the world. These treatments are geared towards combating pulmonary infections, slowing its progression, and minimizing symptoms. These treatments include nebulized therapy, antibiotics, and anti-inflammatories such as oral corticosteroids combined with respiratory and oxygen therapy to help improve lung function. Adding nutritional supplements as well as monitoring diet will help digestion function and glycaemia control in

An overview of the genetic disease Cystic Fibrosis

1275 words - 5 pages common is blockage of pancreatic ducts, restricting delivery of critical digestive enzymes to the bowel. When the scarred pancreas can no longer produce enough insulin, type 1, or insulin-dependent diabetes is the result. People with Cystic Fibrosis also often acquire impaired reproductive function. Among adult men with CF, 98% produce no or little sperm do to abnormal ducts, and 95% are infertile. In women, cervical secretions are too thick

Cystic Fibrosis Transmembrane Conductance Regulator

2479 words - 10 pages fibrosis. Critical Care Nurse, 25 (40), 46-51. http://ccn.aacnjournals.org/content/25/4/46.full Freudenrich, C. (n.d.). How DNA works. Retrieved from http://science.howstuffworks.com/life/cellular-microscopic/dna4.htm National Coalition for Health Professional Education in Genetics (n.d.). Cystic fibrosis. Retrieved from http://www.nchpeg.org/nutrition/index.php?option=com_content&view=article&id=462&Itemid=564&showall=1 Newson, L

An Idiopathic Autoinmune Disease: Sarcoidosis

1557 words - 7 pages Sarcoidosis is an idiopathic autoimmune disease (Page, 2008). In order for doctors to determine exactly what causes this chronic inflammatory disease research is in demand (Culver, 2011). Clinical manifestations of sarcoidosis are widespread but almost always involve the respiratory tract over the course of the disease (Schoppet, Pankuweit & Maisch, 2011). Sarcoidosis can be evident almost immediately but can also go unnoticed over a long

The Teacher That Was Like Father

837 words - 4 pages any friends. I believe fat people like me shouldn’t be happy, I believe they shouldn’t fall in love either. To me fat people should be miserable, like me.’ Mr. Odis looked over to Makenzie and said, ‘We will go deeper into that later, now it’s your turn Jonathan.’ Johnathan began to talk, ‘Well my name is Johnathan Marshal,’ he paused took a deep breath and started back talking, ‘I was born with Idiopathic Pulmonary Fibrosis, that is scarring or

About Horner's Syndrome

1435 words - 6 pages Horner’s syndrome. Conditions such as; apical pulmonary disease, aortic aneurisms, migraines, metastasis, goiter and a multiple of other causes can inhibit signals from the sympathetic pathway to the ocular structures (Cogan, 1948, p.178). Horner’s syndrome can be congenital, in which the damage to sympathetic nervous system is present from birth. The congenital form has a high incidence of heterochromia, in which iris coloration is different

Atrial Fibrillation: Causes, Pathophysiology and Treatment

2016 words - 8 pages conduction or structure of the atria, inflammation, fibrosis or dilation. After a trigger, in a healthy atrium and early in the course of AF, sinus rhythm can be spontaneously restored. The trigger for AF is usually a rapidly firing focus, and the commonest site of origin is the pulmonary veins. This is demonstrated in that during catheter ablation of AF, success is largely dependent on isolating the pulmonary veins electrically. Other less

FGF Signaling and Early Lung Development

1744 words - 7 pages that controls pulmonary development(Kimura and Deutsch, 2007). Namely, FGF-10 and FGF-7 play critical roles in branching morphogenesis. FGF-10 is highly concentrated in the distal lung mesenchyme in a pattern that appears to link with other sites initiating bud formation(Cardoso and Lü, 2006). The development of the lung is highly contested, as the control of growth is not fully understood. It is increasingly clear that in utero conditions

Alcohol Liver Disease

1274 words - 6 pages In addition to the disruption of fatty acid synthesis and oxidation, ethanol alters lipid droplet (LD, the storage form of TG) metabolism in hepatocytes and very low-density lipoprotein secretion from the liver. This effect, together with the inhibition of mitochondrial fatty acid β-oxidation, contributes to the pathogenesis of fatty liver (hepatosteatosis), the initial stage of ALD. Another major pathway of ethanol metabolism that involves

Pathway Reconstruction of Airway Remodeling in Chronic Lung Diseases

2526 words - 10 pages response to wounding [41]. One of the lung disorders that have both inflammatory and remodeling features is mustard lung. Imbalance between levels of MMPs and TIMPs in a particular location can lead to accumulation of ECM protein at that site. The most well-known profibrotic cytokine TGF-β is known to crosstalk with the ECM pathway. Also, TGF-β signaling has a critical role in activation of angiogenic factor [42]. Mucus hyper-secretion Mucus

Similar Essays

The Unknown World Of Idiopathic Pulmonary Fibrosis:

657 words - 3 pages Introduction “Idiopathic pulmonary fibrosis is a specific form of progressive, fibrosis interstitial pneumonia of unknown etiology.”1 More common in older males, IPF has not given up any of its secrets for over 50 years. Despite all the data and research a lot is still unknown, causing little progression in knowledge and patient mortality un-changing. An estimated 30,000 cases of IPF are found in the United States per year with a median survival

Hydro Alcoholic Extract Of Raphanus Sativus L.Var Niger Attenuates Bleomycin Induced Pulmonary Fibrosis Via Decreasing Tgf β1 Level

2792 words - 12 pages J, Brown KK, et al. onbehalf of the ATS/ERS/JRS/ALATCommittee on Idiopathic Pulmonary Fibrosis,2011.Anofficial ATS/ERS/JRS/ALATstatement:idiopathic pulmonary fibrosis:evidence based guide lines for diagnosis and management. American Journal of Respiratory and Critical Care Medicine. 2011;183:788–824. 10. Klingsberg RC, Mutsaers SE, Lasky JA. Curren tclinical trials for the treatment of idiopathic pulmonary fibrosis. Respirology. 2010;15:19-31. 11

Pathology And Treatment Of Pulmonary Arterial Hypertension

2205 words - 9 pages involvement in the pathology of idiopathic pulmonary arterial hypertension. American Journal of Respiratory and Critical Care Medicine, 186(9), 897-908. Tuder, R. M., Abman, S. H., Braun, T., Capron, F., Stevens, T., Thistlewaite, P. A., & Haworth, S. G. (2009, April). Development and pathology of pulmonary hypertension. Journal of the American College of Cardiology, 54(1).

Potential Of Applying The Radial Basis Function

651 words - 3 pages Potential of applying the radial basis function (RBF) neural network architecture for the classification of biological microscopic images displaying lung tissue sections with idiopathic pulmonary fibrosis. For the development of the RBF classifiers, the fuzzy means clustering algorithm is utilized. This method is based on a fuzzy partition of the input space and requires only a short amount of time to select both the structure and the